Most of the symptoms you mention are possibly related to thalassemia, but it is very hard to say for sure without seeing some blood test results for both you and your daughter. Alpha thalassemia carrier status is not easy to diagnose. If your child has serious health issues resulting from alpha thalassemia, it is most likely 3 gene affected alpha thal, which is called Hemoglobin H disease. You can read more about it at the previous link I supplied in answer to your other post and also at
http://www.thalassemia.com/alpha_thal_3.htmlHemoglobin H disease is characterized by one functional gene that codes for the production of alpha globins (--/-a). Hgb H disease should be considered in the case of a neonate in whom all of the red blood cells are very hypochromic. These neonates have a high percentage of hemoglobin Bart's on the their newborn screening results. In older children, this hemoglobinopathy is characterized by moderate anemia with a hemoglobin in the 8 to 10 gm/dL range, hypochromia, microcytosis, red cell fragmentation, and a fast migrating hemoglobin (Hgb H) on electrophoresis.
Hemoglobin H does not function as a normal hemoglobin and has a high oxygen affinity (holds onto oxygen longer making it unavaible for use by the body), so the measured hemoglobin in these children is misleading. Individuals who have Hgb H generally have a persistent stable state of anemia, which may be accentuated by increased hemolysis during viral infections and by exposure to oxidant medications, chemicals and foods such as sulfa drugs, benzene, and fava beans (similar to individuals who have G6PD deficiency). As the red cells mature they loose their ability to withstand oxidant stress and Hgb H precipitates, leading to hemolysis. Therapy for individuals who have Hgb H disease includes folate, avoidance of oxidant drugs and foods, genetic counseling education and frequent medical care. Uncommon occurrences in a child with Hgb H would be severe anemia, cholelithiasis, skin ulceration, and splenomegaly requiring splenectomy. Unlike individuals who have beta thalassemia, hemosiderosis is rare in Hgb H disease.
The main treatment for HbH disease is folic acid and regular medical monitoring. The dosage of folic acid should be 1000-10,000 mcg (micrograms). 1000 mcg = 1 mg.
If your child has HbH disease, then both parents have to be alpha thal carriers, as it is a 3 gene condition, which means 3 of the 4 alpha hemoglobin genes are deleted. Two alpha genes are received from each parent, so both parents have to be carriers for the child to have 3 alpha thal genes.
Please pay special attention to the need for genetic counseling. Also, diet and nutrition can play key roles in maintaining health. See our thread at
http://www.thalassemiapatientsandfriends.com/index.php?topic=118.msg578#msg578