Yes, there will be no HbA, as there is no gene that can produce it. With codon 15, the beta zero is the result of an amino acid substitution and not a deletion, so the gene is there but useless, so one gene has no function due to the codon 15 mutation. In HPFH, the beta gene is fully deleted, and although the mechanics are not perfectly understood, that allows the gamma gene to fully express, as it does before birth, replacing the function of beta globin with gamma globin, which combines with alpha globin to form fetal hemoglobin. In HBPH, this function is typically enough to provide an Hb level that is no worse than thal minor. The HbF is also distributed more evenly in blood cells, providing a more stable condition.
The reason I mentioned delta beta thalassemia, is because they are very similar genetically and sometimes mistaken for each other, but a DNA analysis is able to differentiate between the two. So, as long as this DNA test was accurate, and there is no reason to believe otherwise, as long as it was done at a true lab and not at an online company like 23andme, the results should be viewed with confidence. I think an Hb of 11 at 13 months is also a sign that you are not dealing with any severe phenotype.
To summarize, the codon 15 is a mutation while the presence of HPFH 3 shows that the beta and delta genes are fully deleted in that gene cluster. Function is replaced by the gamma gene. Your child will always have close to 100% HbF. The cluster that contains the codon 15 beta gene still has a functioning delta gene. The globin produced by the delta gene combines with alpha globin to produce HbA2, hence the small percentage of HbA2 present.