Normally, there are 4 alpha globin genes in clusters of two. When there is an alpha triplication, one of these clusters actually has an extra gene, and the amount of alpha globin produced increases. When one also carried beta thalassemia, a less than normal amount of beta globin is produced, so there is an imbalance between the two globins, that together form hemoglobin. This can create the symptoms of beta thalassemia intermedia. Your report shows only the triplicated gene. Supposedly, this causes no issues. However, since there is still an imbalance of globins produced, I would not be surprised if patients in the real world did sometimes experience some symptoms because of the excess alpha globin chains produced.
It may be wise to have a partner tested to make sure there is no thal carrier status in the partner, as this could create the potential of a child with moderate anemia.